The neuro-cognitive, developmental disease of autism, whose symptoms appear during the first three years of a child’s life, have for years been thought to involve a number of genes, but there was no concrete proof.
Now a team at Cold Spring Harbor Laboratory (CSHL) in New York, led by an Israeli doing postdoctoral fellowship work on mice, has for the first time provided functional evidence that inheriting fewer copies of genes on chromosome #16 leads to autism-like features.
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